history
. Three distinctly different patterns
. Avulsion fracture of styloid process
. Transverse fracture of proximal diaphysis (Jones fracture)
. Distal spiral fractures (Ballet fracture)
. Avulsion fractures occur by inversion mechanism.
. Jones fracture common in running, jumping sports
. May report prodrome of pain prior to presentation with Jones fracture,
suggesting stress reaction in bone
. Lateral foot pain with weight-bearing
physical exam
. Tenderness over base of fifthmetatarsal
. Pain with active eversion of foot
. May have pain with passive inversion
. Be sure to examine lateral ankle ligament stability.
studies
. Plain radiographs (AP, lateral, oblique)
. Avulsion type is usually extra-articular
. Three patterns Jones fracture: acute, delayed union, nonunion
. True acute Jones fracture will have sharp fracture line, no intramedullary
sclerosis.
. Delayed union: see some evidence of pre-existing fracture (wider
fracture gap, some intramedullary sclerosis)
. Don’t confuse avulsion type with two sesamoids: os peroneum (in
peroneus logus) and os vesalianum (in peroneus brevis).
. Don’t confuse avulsion type with unfused apophysis at base of fifth
metatarsal (closes age 16).
differential diagnosis
. Stress fracture
. Lateral ankle ligament injury
. Injury to cuboid-metatarsal joint
. Peroneus brevis tendinitis/tenosynovitis
treatment
. Avulsion fracture of styloid process
. Short-leg walking cast 2–3 weeks
. Weight-bear as tolerated
. Even nonunion of this type of fracture can be left alone.
. Symptomatic nonunion (rare): excise fragment, repair peroneus
brevis
. Displaced intra-articular avulsion fracture (rare): consider open
reduction/internal fixation
. Jones fracture
. Acute: nonweight-bearing cast immobilization 6–8 weeks
. Consider surgical treatment in athlete
. Delayed union: consider trial of nonweight-bearing cast, immobilization,
may require surgery
. Nonunion: surgery
. Medullary curettage and inlay bone graft or closed intramedullary
screw fixation with 4.5-mm malleolar screw
. Use largest screw diameter possible.
. Postop: nonweight-bearing cast for 2 weeks, followed by progressive
weight-bearing in hard-soled shoe
. Ballet fracture
. Short-leg walking cast or Camwalker
disposition
N/A
prognosis
. Acute fractures heal in 6–8 weeks.
. Delayed union treated conservatively may take>1 year for full healing.
. Nonunion treated with surgery heals in average 3months.
. Return to sports after surgery: approximately 8 weeks
. Complications of screw fixation
. Screw fracture
. Screw protrusion out of metatarsal
. Tenderness over screw head
. Refracture
caveats and pearls
. These fractures have propensity for nonunion due to poor blood
supply.
. Refracture can occur if return to sports too early.
. Use of foot orthosis recommended for early return to sports
. Plain radiographs may not be sensitive enough to determine healing
. Consider CT or tomograms.
. If refracture occurs after return to sports, consider exchange to
larger screw or use of inlay bone graft.
ACUTE FRACTURES OF THE FIFTH METATARSAL
Tuesday, October 21, 2008Posted by Andrew at 2:08 AM 0 comments
SOUND MIXING
Monday, October 20, 2008Posted by Andrew at 11:51 AM 0 comments
TREATMENTS FOR ANTERIOR CRUCIATE LIGAMENT INJURY
Sunday, October 19, 2008Treatment
. Acute
. Establish diagnosis
. Ice, resolution of swelling, closed-chain quadmotion recovery
. Avoid immobilization
. Physical therapy
. Consider MRI
. Treatment considerations
. Age
. Activity level
. Category I sports (e.g., basketball, football, soccer, volleyball)
. Associated injuries (e.g., meniscus,MCL)
. Amount of pathologic laxity (e.g., Grade 2 or 3 pivot)
. Sports participation
. Expectation
. KT-1000 maximummanual (>6mmSSD, poor prognosis)
. Treatment options
. Nonsurgical
. Activity modificationmandatory
. ACL brace – Low-level activities
. Does not prevent instability
. Reduces severity and frequency of instability
. May require arthroscopy for meniscal pathology
. Surgical: tissue options
. Patellar tendon autograft
. Hamstrings
. Quadriceps tendon
. Allograft
. Irradiated vs. nonirradiated
. Patellar tendon vs. Achilles tendon
Disposition
N/A
Prognosis
. Nonsurgical treatment
. Low probability of high-level sports participation
. High likelihood of recurrent injuries –meniscal, articular
. Subjective satisfaction generally low
. 50% function due to ACL functional brace
. Surgical treatment
. Preferred for most individuals
. Stability success 85–90%
. Patient satisfaction 95%
. Patellar pain testing variable (10–40%)
. Side-to-side functional testing: 40–90% normal
. Return to play
. Nonoperative
. Should be braced
. Usually 4–6 weeks
. Caution that reinjury common
. Reconstructive
. Graft and sport dependent
. 4–6monthsmost surgeons; others recommend 6–9months
. Criteria – Normal stability, motion functional testing within
10–15%
. Complications of surgical treatment
. Infection (1%)
. Deep venous thrombosis, pulmonary embolus (1%)
. Patellar fracture, patellar tendon rupture (1%)
. Loss ofmotor (extension and/or flexion)
. Recurrent laxity (micro/macrotraumatic)
. Reinjury (5–10%) – Time-dependent
. Opposite side ACL injury –More common than graft disruption
Caveats and Pearls
. Suspect ACL injury in athlete with hemarthrosis.
. A patient with a displaced bucket handle meniscal tear (locked
knee) has an ACL injury until proven otherwise.
. “Partial” ACL injuries are extremely uncommon (<5%); athlete has
an ACL-deficient knee until proven otherwise.
. ACL diagnosis is established by history and exam; MRI should not
be used to establish diagnosis.
. ACL reconstructive surgery is not a surgical emergency.
. Postop motion complications reduced with delayed surgical treatment
. Beware of associated MCL injury, which may contribute to delayed
motion recovery.
. Medial meniscal tears more common in chronic ACL knees; lateral
meniscal tearsmore common in acute injuries
. Postoperative ACL bracing controversial
. ACL injuries three to four times more common in females (multifactorial)
. Accelerated rehab programs for patellar tendon graft current standard
. Surgical results similar for patellar tendon vs. hamstring autograft
. Surgical results of single- vs. double-incision arthroscopic techniques
similar
. Current graft choices: patellar tendon autograft, hamstring, quadriceps
tendon, nonirradiated patellar tendon allograft
. Outpatient ACL reconstruction feasible
. Postop continuous passive motion is longer standard of care
. Avoid open-chain quad extension exercises during rehab.
Posted by Andrew at 10:32 AM 0 comments
ACUTE NAVICULAR FRACTURE
Saturday, October 18, 2008
History
. History of trauma
. Midfoot pain and swelling
Physical exam
. Swelling and exquisite pain on dorsomedial aspect of midfoot
. Dorsal lip avulsion – two ligaments insert on dorsum of navicular
. Dorsal talonavicular
. Stressed with inversion and plantarflexion
. Anterior aspect of deltoid ligament
. Stressed with eversion
. Tuberosity fractures
. Result of acute valgus or eversion injury increases stress on posterior
tibialis tendon
Studies
. AP, oblique, and lateral radiographs
. Examine closely formidtarsal joint (Lisfranc) injuries
. Bone scans, CT scan, MRI for occult fractures
. Differentiate acute tuberosity fracture fromaccessory navicular
. Accessory navicular is smooth and regular
Differential diagnosis
. Cuneiformand cuboid fractures
. Navicular stress fracture
. Running or jumping athletes
. Navicular avulsion fracture
Treatment
. Dorsal lip avulsion
. Conservative
. Weight-bearing cast for 4–6 weeks
. Open reduction and internal fixation if fragment is>25% of navicular
. Displaced acute fractures treated with anatomic and stable internal
fixation
. Anatomic reduction of talonavicular joint more critical
. Mobility of this joint is important for function
. Anatomic reduction of anterior and distal navicular not critical
. Naviculocuneiformjoints have littlemotion
Disposition
N/A
Prognosis
. Navicular is largely covered with articular cartilage
. Notmuch room for nutrient vessels to enter
. Makes the tarsal navicular subject to osteonecrosis
Caveats and Pearls
. Located in the uppermost part of the arch, the navicular is the keystone
for vertical stress on the arch
. Anatomic reduction essential to restore talonavicular motion
Posted by Andrew at 8:13 AM 0 comments
LEARNING from the BEST – PART A
Wednesday, October 15, 2008Posted by Andrew at 5:17 AM 0 comments
ANTERIOR CRUCIATE LIGAMENT INJURY
Tuesday, October 14, 2008noncontact, hyperextension
. Patient may recall pop or tearing sensation – 80%
. Painful, inability to continue activity
. Rapid hemarthrosis within 3 hours – 80%
. If chronicmay have history of recurrent instability
. Common sports: skiing, basketball, volleyball, football, soccer
. May be isolated or in conjunction with multiple ligament injuries –
medial collateral (MCL), posterior cruciate (PCL), posterolateral or
knee dislocation
. Often associated with meniscal pathology and/or articular cartilage
injury
. Knee instability complaints: jumping, twisting, deceleration, cutting
activities
Physical exam
.Acute: effusion, bloody hemarthrosis if aspirated
. Joint line tenderness suggestive of associated meniscal pathology
. Ligament laxity exam – compare to uninjured knee
. Abnormal Lachman testmost sensitive test
. Increased anterior tibial translation @ 20–30. knee fusion
. Positive pivot shift test (Losee, Hughston, flexion rotator drawer
variants)
. Pathognomonic of complete anterior cruciate ligament (ACL) injury
. Subluxation – Reduction phenomenon related to axial compression,
valgus loading, and flexion/extension occurred at 15–30.
. Associated ligament laxity tests
. MCL – Valgus laxity at 0., 30.
. Lateral collateral ligament – Valgus laxity at 0., 30.
. PCL – Increased posterior translation at 90. (posterior drawer),
posterior sag test (gravity flexion test)
. Posterolateral – Increased posterolateral rotation, increased
external tibial rotation, asymmetric dial test
. Locked knee –Displaced bucket handle meniscal tear, rule out associated
ACL injury
Studies
. Radiographs
. AP, lateral, tunnel,Merchant (four views)
. Generally normal
. Lateral capsular sign (Segond fracture): marginal avulsion fracture
fromanterolateral tibial plateau pathognomonic
. Lateral notch sign (chronic ACL): accentuation of indentation
of sulcus terminalis in lateral femoral condyle; rarely seen with
PCL/posterolateral corner (PLC) injury
. Chronic ACL deficiency: periarticular osteophytes, tibial eminence
peaking, intercondylar notch narrowing
. MRI
. Highly sensitive/specific for ACL injury
. Generally does not differentiate between partial or complete
ACL injury
. Associated meniscal pathology common
. Bone bruise noted (80%), lateral femoral condyle, lateral tibial
condyle most common
. Effusion frequently noted
. MRI value: detecting associated meniscal pathology and articular
cartilage pathology
. KT-1000
. Instrumented laxity testing device
. Measures side-to-side differences (SSD) and absolute translation
. Dx: maximum manual SSD >3 mm; 30-pound anterior translation
>10mm
Differential diagnosis
. Hemarthrosis
. ACL, patellar dislocation, peripheral meniscal tear, intra-articular
fracture, PCL injury, popliteus tendon avulsion
. Instability
. Patellar instability
. Meniscal tear (e.g., bucket handle)
. Posterolateral
. Quad weakness – giving way with level walking/standing
Posted by Andrew at 12:20 AM 0 comments
ACUTE COMPARTMENT SYNDROME
Sunday, October 12, 2008
history
. Definition: Increased pressure in a confined tissue space that
reduces capillary blood flow below a level necessary for viability of
normal tissue
. Etiology 1: Increased volume within a closed space (i.e., trauma,
hemorrhage, or reperfusion injury)
. Etiology 2: Decreased size of an enclosed space (i.e. cast, constrictive
dressing, orMAST trousers)
Pathogenesis
. Muscle injury: edema/hemorrhage – Increased pressure in enclosed
space – ischemia – further soft tissue damage
. Circulatory injury: Swelling with reperfusion – increased pressure in
enclosed space – ischemia – further soft tissue damage
. Muscle ischemia reversible up to 4 hours, irreversible after 8
. Nerve ischemia results in reversible neuropraxia under 3 hours and
irreversible after 8
Compartments
. Anterior
. Lateral
. Deep posterior
. Superficial posterior
physical exam
. Five P’s: Pain, Pallor, Paresthesias, Pulselessness, Paralysis
. Pain out of proportion to injury
. Pain with passive stretch of foot
. Pallor skin tone
. Loss of or decreased pulses (uncommon and/or a very late finding)
. Paralysis or sensory changes after ischemia >1 hour
. Tense, swollen compartments (most sensitive finding)
. Glossy appearance of skin
studies
. Labs
. Elevated CPK values are common with ischemia but also elevated
in trauma
. Compartment pressuremeasurement
. Indicated in polytrauma, obtunded patient, orwith inconclusive
clinical diagnosis
. Direct measurement of involved compartments using needle
catheter such as a Stryker STIC catheter, WICK catheter, or
transducer fromarterial line
. Pressure threshold requiring fasciotomy is controversial.
. Multiple sampling sites, with the highest value recorded and
used to determine the need for fasciotomy
. Fasciotomy recommended with a measured pressure >35
mmHg or a pressure 20 mmHg below the measured diastolic
blood pressure (number varies)
differential diagnosis
. Compartment syndrome is a surgical emergency. If clinically suspected,
then the diagnosis is compartment syndrome until proven
otherwise.
treatment
. Nonoperative
. Remove compressive dressings, casts, etc.
. Elevate leg to level of heart only.
. Compartment measurements if clinically suspicious
. Operative
. Two-incision fasciotomy to decompress compartments
. Anterolateral incision – half the distance between fibula and tibial
crest; used to decompress the anterior and lateral compartments
. Beware exiting superficial peroneal nerve through fascial defect
distally
. Posteriormedial incision – 2 cmposterior to medial tibia
. Delayed primary closure at 4–7 days with possible skin graft if
Needed
disposition
N/A
prognosis
. Good if recognized and treated early
. Poor if delayed diagnosis and/or intervention
Complications
. Infection
. Claw toes
. Dysfunctional extremity
. Amputation
caveats and pearls
. Early recognition is the key to successful treatment.
. Remember the five P’s.
. If you are thinking about checking the compartment pressures,
check them!
. Acute compartment syndrome is a surgical emergency.
Posted by Andrew at 2:02 AM 0 comments
